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The
cell
biology
of asthma
Erle, David J ; Sheppard, Dean
The Journal of
cell
biology
, 2014-06, Vol.205 (5), p.621-631
[Periódico revisado por pares]
United States: The Rockefeller University Press
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Título:
The
cell
biology
of asthma
Autor:
Erle, David J
;
Sheppard, Dean
Assuntos:
Adrenal Cortex Hormones - therapeutic use
;
Animals
;
Asthma - drug therapy
;
Asthma - metabolism
;
Asthma - physiopathology
;
Cell
Adhesion
;
Cell
Communication
;
Cell
Differentiation
;
Cilia - metabolism
;
Epithelial Cells - cytology
;
Epithelial Cells - pathology
;
Humans
;
Interleukin-13 - metabolism
;
Muscle Contraction - drug effects
;
Myocytes, Smooth Muscle - cytology
;
Myocytes, Smooth Muscle - pathology
;
Myosins - metabolism
;
Receptor, Epidermal Growth Factor - metabolism
;
Receptors, Notch - metabolism
;
Reviews
;
Signal Transduction
;
Th2 Cells - cytology
;
Th2 Cells - metabolism
É parte de:
The Journal of
cell
biology
, 2014-06, Vol.205 (5), p.621-631
Notas:
ObjectType-Article-2
SourceType-Scholarly Journals-1
ObjectType-Feature-3
content type line 23
ObjectType-Review-1
Descrição:
The clinical manifestations of asthma are caused by obstruction of the conducting airways of the lung. Two airway
cell
types are critical for asthma pathogenesis: epithelial cells and smooth muscle cells. Airway epithelial cells, which are the first line of defense against inhaled pathogens and particles, initiate airway inflammation and produce mucus, an important contributor to airway obstruction. The other main cause of airway obstruction is contraction of airway smooth muscle. Complementary experimental approaches involving cultured cells, animal models, and human clinical studies have provided many insights into diverse mechanisms that contribute to airway epithelial and smooth muscle
cell
pathology in this complex disease.
Editor:
United States: The Rockefeller University Press
Idioma:
Inglês
Links
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