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1 |
Material Type: Artigo
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UCP2‐dependent improvement of mitochondrial dynamics protects against acute kidney injuryQin, Nan ; Cai, Ting ; Ke, Qingqing ; Yuan, Qi ; Luo, Jing ; Mao, Xiaoming ; Jiang, Lei ; Cao, Hongdi ; Wen, Ping ; Zen, Ke ; Zhou, Yang ; Yang, JunweiThe Journal of pathology, 2019-03, Vol.247 (3), p.392-405 [Periódico revisado por pares]Chichester, UK: John Wiley & Sons, LtdTexto completo disponível |
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2 |
Material Type: Artigo
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UCP2 ameliorates mitochondrial dysfunction, inflammation, and oxidative stress in lipopolysaccharide-induced acute kidney injuryDing, Yue ; Zheng, Yijun ; Huang, Jinda ; Peng, Wanwan ; Chen, Xinxin ; Kang, Xiangjin ; Zeng, QiyiInternational immunopharmacology, 2019-06, Vol.71, p.336-349 [Periódico revisado por pares]Netherlands: Elsevier B.VTexto completo disponível |
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3 |
Material Type: Artigo
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Mitochondrial uncoupling protein‐2 reprograms metabolism to induce oxidative stress and myofibroblast senescence in age‐associated lung fibrosisRangarajan, Sunad ; Locy, Morgan L. ; Chanda, Diptiman ; Kurundkar, Ashish ; Kurundkar, Deepali ; Larson‐Casey, Jennifer L. ; Londono, Pilar ; Bagchi, Rushita A. ; Deskin, Brian ; Elajaili, Hanan ; Nozik, Eva S. ; Deshane, Jessy S. ; Zmijewski, Jaroslaw W. ; Eickelberg, Oliver ; Thannickal, Victor J.Aging cell, 2022-09, Vol.21 (9), p.e13674-n/a [Periódico revisado por pares]England: John Wiley & Sons, IncTexto completo disponível |
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4 |
Material Type: Artigo
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Melatonin Balance the Autophagy and Apoptosis by Regulating UCP2 in the LPS-Induced CardiomyopathyPan, Pan ; Zhang, Hongmin ; Su, Longxiang ; Wang, Xiaoting ; Liu, DaweiMolecules (Basel, Switzerland), 2018-03, Vol.23 (3), p.675 [Periódico revisado por pares]Switzerland: MDPI AGTexto completo disponível |
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5 |
Material Type: Artigo
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Endothelial Uncoupling Protein 2 Regulates Mitophagy and Pulmonary Hypertension During Intermittent HypoxiaHaslip, Maria ; Dostanic, Iva ; Huang, Yan ; Zhang, Yi ; Russell, Kerry S ; Jurczak, Michael J ; Mannam, Praveen ; Giordano, Frank ; Erzurum, Serpil C ; Lee, Patty JArteriosclerosis, thrombosis, and vascular biology, 2015-05, Vol.35 (5), p.1166-1178 [Periódico revisado por pares]United States: American Heart Association, IncTexto completo disponível |
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6 |
Material Type: Artigo
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A Ketogenic Diet Improves Mitochondrial Biogenesis and Bioenergetics via the PGC1α-SIRT3-UCP2 AxisHasan-Olive, Md Mahdi ; Lauritzen, Knut H. ; Ali, Mohammad ; Rasmussen, Lene Juel ; Storm-Mathisen, Jon ; Bergersen, Linda H.Neurochemical research, 2019-01, Vol.44 (1), p.22-37 [Periódico revisado por pares]New York: Springer USTexto completo disponível |
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7 |
Material Type: Artigo
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Molecular Dynamics Simulations of Mitochondrial Uncoupling Protein 2Škulj, Sanja ; Brkljača, Zlatko ; Kreiter, Jürgen ; Pohl, Elena E ; Vazdar, MarioInternational journal of molecular sciences, 2021-01, Vol.22 (3), p.1214 [Periódico revisado por pares]Switzerland: MDPI AGTexto completo disponível |
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8 |
Material Type: Artigo
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Leishmania donovani inhibits inflammasome‐dependent macrophage activation by exploiting the negative regulatory proteins A20 and UCP2Gupta, Anand Kumar ; Ghosh, Kuntal ; Palit, Shreyasi ; Barua, Jayita ; Das, Pijush K. ; Ukil, AninditaThe FASEB journal, 2017-11, Vol.31 (11), p.5087-5101 [Periódico revisado por pares]United States: Federation of American Societies for Experimental BiologyTexto completo disponível |
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9 |
Material Type: Artigo
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Uncoupling Proteins as Therapeutic Targets for Neurodegenerative DiseasesBarnstable, Colin J ; Zhang, Mingliang ; Tombran-Tink, JoyceInternational journal of molecular sciences, 2022-05, Vol.23 (10), p.5672 [Periódico revisado por pares]Switzerland: MDPI AGTexto completo disponível |
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10 |
Material Type: Artigo
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Genipin inhibits NLRP3 and NLRC4 inflammasome activation via autophagy suppressionYu, Shui-Xing ; Du, Chong-Tao ; Chen, Wei ; Lei, Qian-Qian ; Li, Ning ; Qi, Shuai ; Zhang, Xiao-Jing ; Hu, Gui-Qiu ; Deng, Xu-Ming ; Han, Wen-Yu ; Yang, Yong-JunScientific reports, 2015-12, Vol.5 (1), p.17935-17935, Article 17935 [Periódico revisado por pares]England: Nature Publishing GroupTexto completo disponível |