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Wnt5b‐associated exosomes promote cancer cell migration and proliferation
Wnt5b‐associated exosomes promote cancer cell migration and proliferation
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Wnt5b‐associated exosomes promote cancer cell migration and proliferation

Harada, Takeshi ; Yamamoto, Hideki ; Kishida, Shosei ; Kishida, Michiko ; Awada, Chihiro ; Takao, Toshifumi ; Kikuchi, Akira

Cancer science, 2017-01, Vol.108 (1), p.42-52 [Periódico revisado por pares]

England: John Wiley & Sons, Inc

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2
CKAP4, a DKK1 Receptor, Is a Biomarker in Exosomes Derived from Pancreatic Cancer and a Molecular Target for Therapy
CKAP4, a DKK1 Receptor, Is a Biomarker in Exosomes Derived from Pancreatic Cancer and a Molecular Target for Therapy
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CKAP4, a DKK1 Receptor, Is a Biomarker in Exosomes Derived from Pancreatic Cancer and a Molecular Target for Therapy

Kimura, Hirokazu ; Yamamoto, Hideki ; Harada, Takeshi ; Fumoto, Katsumi ; Osugi, Yoshihito ; Sada, Ryota ; Maehara, Natsumi ; Hikita, Hayato ; Mori, Soichiro ; Eguchi, Hidetoshi ; Ikawa, Masahito ; Takehara, Tetsuo ; Kikuchi, Akira

Clinical cancer research, 2019-03, Vol.25 (6), p.1936-1947 [Periódico revisado por pares]

United States

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3
ARL4C is associated with initiation and progression of lung adenocarcinoma and represents a therapeutic target
ARL4C is associated with initiation and progression of lung adenocarcinoma and represents a therapeutic target
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ARL4C is associated with initiation and progression of lung adenocarcinoma and represents a therapeutic target

Kimura, Kenji ; Matsumoto, Shinji ; Harada, Takeshi ; Morii, Eiichi ; Nagatomo, Izumi ; Shintani, Yasushi ; Kikuchi, Akira

Cancer science, 2020-03, Vol.111 (3), p.951-961 [Periódico revisado por pares]

England: John Wiley & Sons, Inc

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4
Ribonucleotide Reductase Catalytic Subunit M1 (RRM1) as a Novel Therapeutic Target in Multiple Myeloma
Ribonucleotide Reductase Catalytic Subunit M1 (RRM1) as a Novel Therapeutic Target in Multiple Myeloma
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Ribonucleotide Reductase Catalytic Subunit M1 (RRM1) as a Novel Therapeutic Target in Multiple Myeloma

Sagawa, Morihiko ; Ohguchi, Hiroto ; Harada, Takeshi ; Samur, Mehmet K ; Tai, Yu-Tzu ; Munshi, Nikhil C ; Kizaki, Masahiro ; Hideshima, Teru ; Anderson, Kenneth C

Clinical cancer research, 2017-09, Vol.23 (17), p.5225-5237 [Periódico revisado por pares]

United States: American Association for Cancer Research Inc

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5
Targeting histone deacetylase 3 (HDAC3) in the bone marrow microenvironment inhibits multiple myeloma proliferation by modulating exosomes and IL-6 trans-signaling
Targeting histone deacetylase 3 (HDAC3) in the bone marrow microenvironment inhibits multiple myeloma proliferation by modulating exosomes and IL-6 trans-signaling
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Targeting histone deacetylase 3 (HDAC3) in the bone marrow microenvironment inhibits multiple myeloma proliferation by modulating exosomes and IL-6 trans-signaling

Ho, Matthew ; Chen, Tianzeng ; Liu, Jiye ; Dowling, Paul ; Hideshima, Teru ; Zhang, Li ; Morelli, Eugenio ; Camci-Unal, Gulden ; Wu, Xinchen ; Tai, Yu-Tzu ; Wen, Kenneth ; Samur, Mehmet ; Schlossman, Robert L ; Mazitschek, Ralph ; Kavanagh, Emma L ; Lindsay, Sinéad ; Harada, Takeshi ; McCann, Amanda ; Anderson, Kenneth C ; O'Gorman, Peter ; Bianchi, Giada

Leukemia, 2020-01, Vol.34 (1), p.196-209 [Periódico revisado por pares]

England: Nature Publishing Group

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6
TAK1 is a pivotal therapeutic target for tumor progression and bone destruction in myeloma
TAK1 is a pivotal therapeutic target for tumor progression and bone destruction in myeloma
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TAK1 is a pivotal therapeutic target for tumor progression and bone destruction in myeloma

Teramachi, Jumpei ; Tenshin, Hirofumi ; Hiasa, Masahiro ; Oda, Asuka ; Bat-Erdene, Ariunzaya ; Harada, Takeshi ; Nakamura, Shingen ; Ashtar, Mohannad ; Shimizu, So ; Iwasa, Masami ; Sogabe, Kimiko ; Oura, Masahiro ; Fujii, Shiro ; Kagawa, Kumiko ; Miki, Hirokazu ; Endo, Itsuro ; Haneji, Tatsuji ; Matsumoto, Toshio ; Abe, Masahiro

Haematologica (Roma), 2021-05, Vol.106 (5), p.1401-1413 [Periódico revisado por pares]

Italy: Fondazione Ferrata Storti

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7
Chemically Modified Antisense Oligonucleotide Against ARL4C Inhibits Primary and Metastatic Liver Tumor Growth
Chemically Modified Antisense Oligonucleotide Against ARL4C Inhibits Primary and Metastatic Liver Tumor Growth
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Chemically Modified Antisense Oligonucleotide Against ARL4C Inhibits Primary and Metastatic Liver Tumor Growth

Harada, Takeshi ; Matsumoto, Shinji ; Hirota, Suguru ; Kimura, Hirokazu ; Fujii, Shinsuke ; Kasahara, Yuuya ; Gon, Hidetoshi ; Yoshida, Toshihiko ; Itoh, Tomoo ; Haraguchi, Naotsugu ; Mizushima, Tsunekazu ; Noda, Takehiro ; Eguchi, Hidetoshi ; Nojima, Satoshi ; Morii, Eiichi ; Fukumoto, Takumi ; Obika, Satoshi ; Kikuchi, Akira

Molecular cancer therapeutics, 2019-03, Vol.18 (3), p.602-612 [Periódico revisado por pares]

United States

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8
Tgf-Beta inhibition restores terminal osteoblast differentiation to suppress myeloma growth
Tgf-Beta inhibition restores terminal osteoblast differentiation to suppress myeloma growth
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Tgf-Beta inhibition restores terminal osteoblast differentiation to suppress myeloma growth

Takeuchi, Kyoko ; Abe, Masahiro ; Hiasa, Masahiro ; Oda, Asuka ; Amou, Hiroe ; Kido, Shinsuke ; Harada, Takeshi ; Tanaka, Osamu ; Miki, Hirokazu ; Nakamura, Shingen ; Nakano, Ayako ; Kagawa, Kumiko ; Yata, Kenichiro ; Ozaki, Shuji ; Matsumoto, Toshio Mei, Lin

PloS one, 2010-03, Vol.5 (3), p.e9870 [Periódico revisado por pares]

United States: Public Library of Science

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9
Cdc42 Is Critical for Cartilage Development During Endochondral Ossification
Cdc42 Is Critical for Cartilage Development During Endochondral Ossification
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Cdc42 Is Critical for Cartilage Development During Endochondral Ossification

Suzuki, Wataru ; Yamada, Atsushi ; Aizawa, Ryo ; Suzuki, Dai ; Kassai, Hidetoshi ; Harada, Takeshi ; Nakayama, Mutsuko ; Nagahama, Ryo ; Maki, Koutaro ; Takeda, Shu ; Yamamoto, Matsuo ; Aiba, Atsu ; Baba, Kazuyoshi ; Kamijo, Ryutaro

Endocrinology (Philadelphia), 2015-01, Vol.156 (1), p.314-322 [Periódico revisado por pares]

United States: Endocrine Society

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10
Activating transcription factor 4, an ER stress mediator, is required for, but excessive ER stress suppresses osteoblastogenesis by bortezomib
Activating transcription factor 4, an ER stress mediator, is required for, but excessive ER stress suppresses osteoblastogenesis by bortezomib
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Activating transcription factor 4, an ER stress mediator, is required for, but excessive ER stress suppresses osteoblastogenesis by bortezomib

Nakamura, Shingen ; Miki, Hirokazu ; Kido, Shinsuke ; Nakano, Ayako ; Hiasa, Masahiro ; Oda, Asuka ; Amou, Hiroe ; Watanabe, Keiichiro ; Harada, Takeshi ; Fujii, Shiro ; Takeuchi, Kyoko ; Kagawa, Kumiko ; Ozaki, Shuji ; Matsumoto, Toshio ; Abe, Masahiro

International journal of hematology, 2013-07, Vol.98 (1), p.66-73 [Periódico revisado por pares]

Tokyo: Springer Japan

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