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1
NF-κB is a critical mediator of BRCA1-induced chemoresistance
NF-κB is a critical mediator of BRCA1-induced chemoresistance
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NF-κB is a critical mediator of BRCA1-induced chemoresistance

Harte, Mary T ; Gorski, Julia J ; Savage, Kienan I ; Purcell, James W ; Barros, Eliana M ; Burn, Philip M ; McFarlane, Cheryl ; Mullan, Paul B ; Kennedy, Richard D ; Perkins, Neil D ; Harkin, D Paul

Oncogene, 2014-02, Vol.33 (6), p.713-723 [Periódico revisado por pares]

England: Nature Publishing Group

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2
T-box 2 represses NDRG1 through an EGR1-dependent mechanism to drive the proliferation of breast cancer cells
T-box 2 represses NDRG1 through an EGR1-dependent mechanism to drive the proliferation of breast cancer cells
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T-box 2 represses NDRG1 through an EGR1-dependent mechanism to drive the proliferation of breast cancer cells

REDMOND, K. L ; CRAWFORD, N. T ; FARMER, H ; D'COSTA, Z. C ; O'BRIEN, G. J ; BUCKLEY, N. E ; KENNEDY, R. D ; JOHNSTON, P. G ; HARKIN, D. P ; MULLAN, P. B

Oncogene, 2010-06, Vol.29 (22), p.3252-3262 [Periódico revisado por pares]

Basingstoke: Nature Publishing Group

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3
Modulation of p53 and MDM2 activity by novel interaction with Ras-GAP binding proteins (G3BP)
Modulation of p53 and MDM2 activity by novel interaction with Ras-GAP binding proteins (G3BP)
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Modulation of p53 and MDM2 activity by novel interaction with Ras-GAP binding proteins (G3BP)

KIM, M. M ; WIEDERSCHAIN, D ; KENNEDY, D ; HANSEN, E ; YUAN, Z.-M

Oncogene, 2007-06, Vol.26 (29), p.4209-4215 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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4
The 2,5 oligoadenylate synthetase RNaseL pathway is a novel effector of BRCA1- and interferon-γ-mediated apoptosis
The 2,5 oligoadenylate synthetase RNaseL pathway is a novel effector of BRCA1- and interferon-γ-mediated apoptosis
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The 2,5 oligoadenylate synthetase RNaseL pathway is a novel effector of BRCA1- and interferon-γ-mediated apoptosis

Mullan, Paul B ; Hosey, Alison M ; Buckley, Niamh E ; Quinn, Jennifer E ; Kennedy, Richard D ; Johnston, Patrick G ; Harkin, D Paul

Oncogene, 2005-08, Vol.24 (35), p.5492-5501 [Periódico revisado por pares]

England: Nature Publishing Group

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5
BRCA1 and GATA3 corepress FOXC1 to inhibit the pathogenesis of basal-like breast cancers
BRCA1 and GATA3 corepress FOXC1 to inhibit the pathogenesis of basal-like breast cancers
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BRCA1 and GATA3 corepress FOXC1 to inhibit the pathogenesis of basal-like breast cancers

Tkocz, D ; Crawford, N T ; Buckley, N E ; Berry, F B ; Kennedy, R D ; Gorski, J J ; Harkin, D P ; Mullan, P B

Oncogene, 2012-08, Vol.31 (32), p.3667-3678 [Periódico revisado por pares]

England: Nature Publishing Group

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6
NF-[kappa]B is a critical mediator of BRCA1-induced chemoresistance
NF-[kappa]B is a critical mediator of BRCA1-induced chemoresistance
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NF-[kappa]B is a critical mediator of BRCA1-induced chemoresistance

Harte, M T ; Gorski, J J ; Savage, K I ; Purcell, J W ; Barros, E M ; Burn, P M ; Mcfarlane, C ; Mullan, P B ; Kennedy, R D ; Perkins, N D ; Harkin, D P

Oncogene, 2014-02, Vol.33 (6), p.713 [Periódico revisado por pares]

New York: Nature Publishing Group

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7
The 2,5 oligoadenylate synthetase/RNaseL pathway is a novel effector of BRCA1- and interferon-[gamma]-mediated apoptosis
The 2,5 oligoadenylate synthetase/RNaseL pathway is a novel effector of BRCA1- and interferon-[gamma]-mediated apoptosis
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The 2,5 oligoadenylate synthetase/RNaseL pathway is a novel effector of BRCA1- and interferon-[gamma]-mediated apoptosis

Mullan, Paul B ; Hosey, Alison M ; Buckley, Niamh E ; Quinn, Jennifer E ; Kennedy, Richard D ; Johnston, Patrick G ; Harkin, D Paul

Oncogene, 2005-08, Vol.24 (35), p.5492 [Periódico revisado por pares]

New York: Nature Publishing Group

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8
The 2,5 oligoadenylate synthetase/RNaseL pathway is a novel effector of BRCA1- and interferon -y -mediated apoptosis
The 2,5 oligoadenylate synthetase/RNaseL pathway is a novel effector of BRCA1- and interferon -y -mediated apoptosis
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The 2,5 oligoadenylate synthetase/RNaseL pathway is a novel effector of BRCA1- and interferon -y -mediated apoptosis

MULLAN, Paul B ; HOSEY, Alison M ; BUCKLEY, Niamh E ; QUINN, Jennifer E ; KENNEDY, Richard D ; JOHNSTON, Patrick G ; HARKIN, D. Paul

Oncogene, 2005, Vol.24 (35), p.5492-5501 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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9
Mutant fibroblast growth factor receptor 3 induces intracellular signaling and cellular transformation in a cell type- and mutation-specific manner
Mutant fibroblast growth factor receptor 3 induces intracellular signaling and cellular transformation in a cell type- and mutation-specific manner
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Mutant fibroblast growth factor receptor 3 induces intracellular signaling and cellular transformation in a cell type- and mutation-specific manner

DI MARTINO, E ; L'HOTE, C. G ; KENNEDY, W ; TOMLINSON, D. C ; KNOWLES, M. A

Oncogene, 2009-12, Vol.28 (48), p.4306-4316 [Periódico revisado por pares]

Basingstoke: Nature Publishing Group

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10
Prolactin induces ERα-positive and ERα-negative mammary cancer in transgenic mice
Prolactin induces ERα-positive and ERα-negative mammary cancer in transgenic mice
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Prolactin induces ERα-positive and ERα-negative mammary cancer in transgenic mice

ROSE-HELLEKANT, Teresa A ; ARENDT, Lisa M ; SCHROEDER, Matthew D ; GILCHRIST, Kennedy ; SANDGREN, Eric P ; SCHULER, Linda A

Oncogene, 2003-07, Vol.22 (30), p.4664-4674 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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