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Refinado por: assunto: Drug Resistance, Neoplasm remover Base de dados/Biblioteca: Springer Nature - Connect here FIRST to enable access remover Nome da Publicação: Leukemia remover
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1
Membrane microparticles mediate transfer of P-glycoprotein to drug sensitive cancer cells
Membrane microparticles mediate transfer of P-glycoprotein to drug sensitive cancer cells
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Membrane microparticles mediate transfer of P-glycoprotein to drug sensitive cancer cells

BEBAWY, M ; COMBES, V ; LEE, E ; JAISWAL, R ; GONG, J ; BONHOURE, A ; GRAU, Ger

Leukemia, 2009-09, Vol.23 (9), p.1643-1649 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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2
Treatment resistance in diffuse large B-cell lymphoma
Treatment resistance in diffuse large B-cell lymphoma
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Treatment resistance in diffuse large B-cell lymphoma

He, Michael Y ; Kridel, Robert

Leukemia, 2021-08, Vol.35 (8), p.2151-2165 [Periódico revisado por pares]

England: Nature Publishing Group

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3
FLT3 D835 mutations confer differential resistance to type II FLT3 inhibitors
FLT3 D835 mutations confer differential resistance to type II FLT3 inhibitors
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FLT3 D835 mutations confer differential resistance to type II FLT3 inhibitors

Smith, C C ; Lin, K ; Stecula, A ; Sali, A ; Shah, N P

Leukemia, 2015-12, Vol.29 (12), p.2390-2392 [Periódico revisado por pares]

England: Nature Publishing Group

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4
Receptor tyrosine kinase Axl is required for resistance of leukemic cells to FLT3-targeted therapy in acute myeloid leukemia
Receptor tyrosine kinase Axl is required for resistance of leukemic cells to FLT3-targeted therapy in acute myeloid leukemia
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Receptor tyrosine kinase Axl is required for resistance of leukemic cells to FLT3-targeted therapy in acute myeloid leukemia

Park, I-K ; Mundy-Bosse, B ; Whitman, S P ; Zhang, X ; Warner, S L ; Bearss, D J ; Blum, W ; Marcucci, G ; Caligiuri, M A

Leukemia, 2015-12, Vol.29 (12), p.2382-2389 [Periódico revisado por pares]

England: Nature Publishing Group

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5
Resistance prediction in AML: analysis of 4601 patients from MRC/NCRI, HOVON/SAKK, SWOG and MD Anderson Cancer Center
Resistance prediction in AML: analysis of 4601 patients from MRC/NCRI, HOVON/SAKK, SWOG and MD Anderson Cancer Center
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Resistance prediction in AML: analysis of 4601 patients from MRC/NCRI, HOVON/SAKK, SWOG and MD Anderson Cancer Center

Walter, R B ; Othus, M ; Burnett, A K ; Löwenberg, B ; Kantarjian, H M ; Ossenkoppele, G J ; Hills, R K ; Ravandi, F ; Pabst, T ; Evans, A ; Pierce, S R ; Vekemans, M-C ; Appelbaum, F R ; Estey, E H

Leukemia, 2015-02, Vol.29 (2), p.312-320 [Periódico revisado por pares]

England: Nature Publishing Group

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6
HMGB1-induced autophagy promotes chemotherapy resistance in leukemia cells
HMGB1-induced autophagy promotes chemotherapy resistance in leukemia cells
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HMGB1-induced autophagy promotes chemotherapy resistance in leukemia cells

LIU, L ; YANG, M ; TANG, D ; CAO, L ; KANG, R ; WANG, Z ; ZHAO, Y ; YU, Y ; XIE, M ; YIN, X ; LIVESEY, K. M ; LOTZE, M. T

Leukemia, 2011-01, Vol.25 (1), p.23-31 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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7
Mutations in CRBN and other cereblon pathway genes are infrequently associated with acquired resistance to immunomodulatory drugs
Mutations in CRBN and other cereblon pathway genes are infrequently associated with acquired resistance to immunomodulatory drugs
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Mutations in CRBN and other cereblon pathway genes are infrequently associated with acquired resistance to immunomodulatory drugs

Jones, J R ; Barber, A ; Le Bihan, Y-V ; Weinhold, N ; Ashby, C ; Walker, B A ; Wardell, C P ; Wang, H ; Kaiser, M F ; Jackson, G H ; Davies, F E ; Chopra, R ; Morgan, G J ; Pawlyn, C

Leukemia, 2021-10, Vol.35 (10), p.3017-3020 [Periódico revisado por pares]

England: Nature Publishing Group

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8
MAPK-ERK is a central pathway in T-cell acute lymphoblastic leukemia that drives steroid resistance
MAPK-ERK is a central pathway in T-cell acute lymphoblastic leukemia that drives steroid resistance
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MAPK-ERK is a central pathway in T-cell acute lymphoblastic leukemia that drives steroid resistance

van der Zwet, Jordy C G ; Buijs-Gladdines, Jessica G C A M ; Cordo', Valentina ; Debets, Donna O ; Smits, Willem K ; Chen, Zhongli ; Dylus, Jelle ; Zaman, Guido J R ; Altelaar, Maarten ; Oshima, Koichi ; Bornhauser, Beat ; Bourquin, Jean-Pierre ; Cools, Jan ; Ferrando, Adolfo A ; Vormoor, Josef ; Pieters, Rob ; Vormoor, Britta ; Meijerink, Jules P P

Leukemia, 2021-12, Vol.35 (12), p.3394-3405 [Periódico revisado por pares]

England: Nature Publishing Group

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9
B7-H3 promotes multiple myeloma cell survival and proliferation by ROS-dependent activation of Src/STAT3 and c-Cbl-mediated degradation of SOCS3
B7-H3 promotes multiple myeloma cell survival and proliferation by ROS-dependent activation of Src/STAT3 and c-Cbl-mediated degradation of SOCS3
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B7-H3 promotes multiple myeloma cell survival and proliferation by ROS-dependent activation of Src/STAT3 and c-Cbl-mediated degradation of SOCS3

Lin, Liang ; Cao, Li ; Liu, Yang ; Wang, Ke ; Zhang, Xinwei ; Qin, Xiaodan ; Zhao, Dandan ; Hao, Jie ; Chang, Yingjun ; Huang, Xiaojun ; Liu, Bei ; Zhang, Jun ; Lu, Jin ; Ge, Qing

Leukemia, 2019-06, Vol.33 (6), p.1475-1486 [Periódico revisado por pares]

England: Nature Publishing Group

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10
Loss of glucocorticoid receptor expression mediates in vivo dexamethasone resistance in T-cell acute lymphoblastic leukemia
Loss of glucocorticoid receptor expression mediates in vivo dexamethasone resistance in T-cell acute lymphoblastic leukemia
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Loss of glucocorticoid receptor expression mediates in vivo dexamethasone resistance in T-cell acute lymphoblastic leukemia

Wandler, Anica M ; Huang, Benjamin J ; Craig, Jeffrey W ; Hayes, Kathryn ; Yan, Hannah ; Meyer, Lauren K ; Scacchetti, Alessandro ; Monsalve, Gabriela ; Dail, Monique ; Li, Qing ; Wong, Jasmine C ; Weinberg, Olga ; Hasserjian, Robert P ; Kogan, Scott C ; Jonsson, Philip ; Yamamoto, Keith ; Sampath, Deepak ; Nakitandwe, Joy ; Downing, James R ; Zhang, Jinghui ; Aster, Jon C ; Taylor, Barry S ; Shannon, Kevin

Leukemia, 2020-08, Vol.34 (8), p.2025-2037 [Periódico revisado por pares]

England: Nature Publishing Group

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