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1
Expression of innate immune defence genes in healthy and onychomycotic nail and stratum corneum
Expression of innate immune defence genes in healthy and onychomycotic nail and stratum corneum
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Expression of innate immune defence genes in healthy and onychomycotic nail and stratum corneum

Mercer, D.K. ; Sairi, T. ; Sroka, E. ; Lamont, H. ; Lawrie, Y. ; O'Neil, D.A.

British journal of dermatology (1951), 2017-07, Vol.177 (1), p.279-281 [Periódico revisado por pares]

England: Oxford University Press

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2
Atrophic acne scar: a process from altered metabolism of elastic fibres and collagen fibres based on transforming growth factor‐β1 signalling
Atrophic acne scar: a process from altered metabolism of elastic fibres and collagen fibres based on transforming growth factor‐β1 signalling
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Atrophic acne scar: a process from altered metabolism of elastic fibres and collagen fibres based on transforming growth factor‐β1 signalling

Moon, J. ; Yoon, J.Y. ; Yang, J.H. ; Kwon, H.H. ; Min, S. ; Suh, D.H.

British journal of dermatology (1951), 2019-12, Vol.181 (6), p.1226-1237 [Periódico revisado por pares]

England: Oxford University Press

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3
Molecular evidence that oral supplementation with lycopene or lutein protects human skin against ultraviolet radiation: results from a double‐blinded, placebo‐controlled, crossover study
Molecular evidence that oral supplementation with lycopene or lutein protects human skin against ultraviolet radiation: results from a double‐blinded, placebo‐controlled, crossover study
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Molecular evidence that oral supplementation with lycopene or lutein protects human skin against ultraviolet radiation: results from a double‐blinded, placebo‐controlled, crossover study

Grether‐Beck, S. ; Marini, A. ; Jaenicke, T. ; Stahl, W. ; Krutmann, J.

British journal of dermatology (1951), 2017-05, Vol.176 (5), p.1231-1240 [Periódico revisado por pares]

England: Oxford University Press

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4
Lipocalin‐2 is expressed by activated granulocytes and keratinocytes in affected skin and reflects disease activity in acne inversa/hidradenitis suppurativa
Lipocalin‐2 is expressed by activated granulocytes and keratinocytes in affected skin and reflects disease activity in acne inversa/hidradenitis suppurativa
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Lipocalin‐2 is expressed by activated granulocytes and keratinocytes in affected skin and reflects disease activity in acne inversa/hidradenitis suppurativa

Wolk, K. ; Wenzel, J. ; Tsaousi, A. ; Witte‐Händel, E. ; Babel, N. ; Zelenak, C. ; Volk, H.‐D. ; Sterry, W. ; Schneider‐Burrus, S. ; Sabat, R.

British journal of dermatology (1951), 2017-11, Vol.177 (5), p.1385-1393 [Periódico revisado por pares]

England: Oxford University Press

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5
Response to Tembhre et al.: ‘Enhanced expression of angiotensin‐converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon‐γ: implication of inflammatory milieu in skin tropism of SARS‐CoV‐2’
Response to Tembhre et al.: ‘Enhanced expression of angiotensin‐converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon‐γ: implication of inflammatory milieu in skin tropism of SARS‐CoV‐2’
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Response to Tembhre et al.: ‘Enhanced expression of angiotensin‐converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon‐γ: implication of inflammatory milieu in skin tropism of SARS‐CoV‐2’

Gehlhausen, J.R. ; Ko, C.J. ; Damsky, W.

British journal of dermatology (1951), 2021-05, Vol.184 (5), p.984-984 [Periódico revisado por pares]

England: Oxford University Press

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6
Enhanced expression of angiotensin‐converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon‐γ: implication of inflammatory milieu in skin tropism of SARS‐CoV‐2
Enhanced expression of angiotensin‐converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon‐γ: implication of inflammatory milieu in skin tropism of SARS‐CoV‐2
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Enhanced expression of angiotensin‐converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon‐γ: implication of inflammatory milieu in skin tropism of SARS‐CoV‐2

Tembhre, M.K. ; Parihar, A.S. ; Sharma, V.K. ; Imran, S. ; Bhari, N. ; Lakshmy, R. ; Bhalla, A.

British journal of dermatology (1951), 2021-03, Vol.184 (3), p.577-579 [Periódico revisado por pares]

England: Oxford University Press

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7
MicroRNA profiling reveals differentially expressed microRNA signatures from the skin of patients with nonsegmental vitiligo
MicroRNA profiling reveals differentially expressed microRNA signatures from the skin of patients with nonsegmental vitiligo
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MicroRNA profiling reveals differentially expressed microRNA signatures from the skin of patients with nonsegmental vitiligo

Mansuri, M.S. ; Singh, M. ; Dwivedi, M. ; Laddha, N.C. ; Marfatia, Y.S. ; Begum, R.

British journal of dermatology (1951), 2014-11, Vol.171 (5), p.1263-1267 [Periódico revisado por pares]

England: Blackwell Publishing Ltd

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8
Hormonal, stem cell and Notch signalling as possible mechanisms of disease in hidradenitis suppurativa: a systems‐level transcriptomic analysis
Hormonal, stem cell and Notch signalling as possible mechanisms of disease in hidradenitis suppurativa: a systems‐level transcriptomic analysis
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Hormonal, stem cell and Notch signalling as possible mechanisms of disease in hidradenitis suppurativa: a systems‐level transcriptomic analysis

Gauntner, Timothy D.

British journal of dermatology (1951), 2019-01, Vol.180 (1), p.203-204 [Periódico revisado por pares]

England: Oxford University Press

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9
Site-specific immunophenotyping of keloid disease demonstrates immune upregulation and the presence of lymphoid aggregates
Site-specific immunophenotyping of keloid disease demonstrates immune upregulation and the presence of lymphoid aggregates
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Site-specific immunophenotyping of keloid disease demonstrates immune upregulation and the presence of lymphoid aggregates

Bagabir, R. ; Byers, R.J. ; Chaudhry, I.H. ; Müller, W. ; Paus, R. ; Bayat, A.

British journal of dermatology (1951), 2012-11, Vol.167 (5), p.1053-1066 [Periódico revisado por pares]

Oxford, UK: Blackwell Publishing Ltd

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10
T helper (Th)1, Th17 and Th2 imbalance in mesenchymal stem cells of adult patients with atopic dermatitis: at the origin of the problem
T helper (Th)1, Th17 and Th2 imbalance in mesenchymal stem cells of adult patients with atopic dermatitis: at the origin of the problem
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T helper (Th)1, Th17 and Th2 imbalance in mesenchymal stem cells of adult patients with atopic dermatitis: at the origin of the problem

Orciani, M. ; Campanati, A. ; Caffarini, M. ; Ganzetti, G. ; Consales, V. ; Lucarini, G. ; Offidani, A. ; Di Primio, R.

British journal of dermatology (1951), 2017-06, Vol.176 (6), p.1569-1576 [Periódico revisado por pares]

England: Oxford University Press

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