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Refinado por: autor: Wang, T remover autor: Giebisch, G remover autor: Amorim, J remover
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Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
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Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet

M. A. Bailey A Cantone; Q Yan; G. G MacGregor; Q Leng; J. B. O Amorim; T Wang; S. C Hebert; G Giebisch; Gerhard Malnic

Kidney International New York v. 70, n. 1, p. 51-59, 2006

New York 2006

Item não circula. Consulte sua biblioteca.(Acessar)

2
Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet
Material Type:
Artigo 		Adicionar ao Meu Espaço

Maxi-K channels contribute to urinary potassium excretion in the ROMK-deficient mouse model of Type II Bartter's syndrome and in adaptation to a high-K diet

M. A. Bailey A Cantone; Q Yan; G. G MacGregor; Q Leng; J. B. O Amorim; T Wang; S. C Hebert; G Giebisch; Gerhard Malnic

Kidney International New York v. 70, n. 1, p. 51-59, 2006

New York 2006

Item não circula. Consulte sua biblioteca.(Acessar)

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Deste Autor:

  1. Malnic, G
  2. Wang, T
  3. Cantone, A
  4. Hebert, S
  5. Yan, Q

Neste Assunto:

  1. Fisiologia

  • Realização: ABCD-USP USP   Logos de Redes Sociais: Freepik

Buscando em bases de dados remotas. Favor aguardar.