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1
1-40 β-amyloid protein fragment modulates the expression of CD44 and CD71 on the astrocytoma cell line in the presence of IL1β and TNFα
1-40 β-amyloid protein fragment modulates the expression of CD44 and CD71 on the astrocytoma cell line in the presence of IL1β and TNFα
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1-40 β-amyloid protein fragment modulates the expression of CD44 and CD71 on the astrocytoma cell line in the presence of IL1β and TNFα

Speciale, Livianna ; Ruzzante, Stefania ; Calabrese, Elena ; Saresella, Marina ; Taramelli, Donatella ; Mariani, Claudio ; Bava, Laura ; Longhi, Renato ; Ferrante, Pasquale

Journal of cellular physiology, 2003-07, Vol.196 (1), p.190-195 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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2
1-Benzyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neurotoxic Compound, Disturbs the Behavioral and Biochemical Effects of l-DOPA: In Vivo and Ex Vivo Studies in the Rat
1-Benzyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neurotoxic Compound, Disturbs the Behavioral and Biochemical Effects of l-DOPA: In Vivo and Ex Vivo Studies in the Rat
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1-Benzyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neurotoxic Compound, Disturbs the Behavioral and Biochemical Effects of l-DOPA: In Vivo and Ex Vivo Studies in the Rat

Wąsik, Agnieszka ; Romańska, Irena ; Michaluk, Jerzy ; Kajta, Małgorzata ; Antkiewicz-Michaluk, Lucyna

Neurotoxicity research, 2014-10, Vol.26 (3), p.240-254 [Periódico revisado por pares]

Boston: Springer US

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3
1-Benzyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Parkinsonism-Inducing Toxin, Strongly Potentiates MAO-Dependent Dopamine Oxidation and Impairs Dopamine Release: Ex vivo and In vivo Neurochemical Studies
1-Benzyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Parkinsonism-Inducing Toxin, Strongly Potentiates MAO-Dependent Dopamine Oxidation and Impairs Dopamine Release: Ex vivo and In vivo Neurochemical Studies
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1-Benzyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Parkinsonism-Inducing Toxin, Strongly Potentiates MAO-Dependent Dopamine Oxidation and Impairs Dopamine Release: Ex vivo and In vivo Neurochemical Studies

Wąsik, Agnieszka ; Romańska, Irena ; Antkiewicz-Michaluk, Lucyna

Neurotoxicity research, 2009, Vol.15 (1), p.15-23 [Periódico revisado por pares]

New York: Springer-Verlag

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4
1‐Methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine Pretreatment Attenuates Methamphetamine‐Induced Dopamine Toxicity
1‐Methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine Pretreatment Attenuates Methamphetamine‐Induced Dopamine Toxicity
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1‐Methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine Pretreatment Attenuates Methamphetamine‐Induced Dopamine Toxicity

Kita, Taizo ; Saraya, Tutomu ; Konishi, Noboru ; Matsunari, Yasunori ; Shimada, Keiji ; Nakamura, Mitsutoshi ; O'Hara, Kiichi ; Wagner, George C. ; Nakashima, Toshikatsu

Pharmacology & toxicology, 2003-02, Vol.92 (2), p.71-80

Oxford, UK: Munksgaard International Publishers

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5
1-methyl-4-phenylpyridinium neurotoxicity is attenuated by adenoviral gene transfer of human Cu/Zn superoxide dismutase
1-methyl-4-phenylpyridinium neurotoxicity is attenuated by adenoviral gene transfer of human Cu/Zn superoxide dismutase
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1-methyl-4-phenylpyridinium neurotoxicity is attenuated by adenoviral gene transfer of human Cu/Zn superoxide dismutase

Barkats, Martine ; Horellou, Philippe ; Colin, Philippe ; Millecamps, Stéphanie ; Faucon-Biguet, Nicole ; Mallet, Jacques

Journal of neuroscience research, 2006-02, Vol.83 (2), p.233-242 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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6
1-Octen-3-ol is formed from its primeveroside after mechanical wounding of soybean leaves
1-Octen-3-ol is formed from its primeveroside after mechanical wounding of soybean leaves
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1-Octen-3-ol is formed from its primeveroside after mechanical wounding of soybean leaves

Ntoruru, Juliano Mwenda ; Ohnishi, Toshiyuki ; Katsumata, Fumiya ; Koeduka, Takao ; Matsui, Kenji

Plant molecular biology, 2022-07, Vol.109 (4-5), p.551-561 [Periódico revisado por pares]

Dordrecht: Springer Netherlands

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7
1 or 2 cm Margins of Excision for T2 Melanomas: Do They Impact Recurrence or Survival?
1 or 2 cm Margins of Excision for T2 Melanomas: Do They Impact Recurrence or Survival?
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1 or 2 cm Margins of Excision for T2 Melanomas: Do They Impact Recurrence or Survival?

Hudson, Laura E. ; Maithel, Shishir K. ; Carlson, Grant W. ; Rizzo, Monica ; Murray, Douglas R. ; Hestley, Andrea C. ; Delman, Keith A.

Annals of surgical oncology, 2013, Vol.20 (1), p.346-351 [Periódico revisado por pares]

New York: Springer-Verlag

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8
1,25-Dihydroxyvitamin D₃ acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis
1,25-Dihydroxyvitamin D₃ acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis
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1,25-Dihydroxyvitamin D₃ acts directly on the T lymphocyte vitamin D receptor to inhibit experimental autoimmune encephalomyelitis

Mayne, Christopher G ; Spanier, Justin A ; Relland, Lance M ; Williams, Calvin B ; Hayes, Colleen E

European journal of immunology, 2011-03, Vol.41 (3), p.822-832 [Periódico revisado por pares]

Weinheim: Wiley-VCH Verlag

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9
1,25-dihydroxyvitamin D3 ameliorates lupus nephritis through inhibiting the NF-κB and MAPK signalling pathways in MRL/lpr mice
1,25-dihydroxyvitamin D3 ameliorates lupus nephritis through inhibiting the NF-κB and MAPK signalling pathways in MRL/lpr mice
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1,25-dihydroxyvitamin D3 ameliorates lupus nephritis through inhibiting the NF-κB and MAPK signalling pathways in MRL/lpr mice

Li, Xuewei ; Liu, Jie ; Zhao, Yingzhe ; Xu, Ning ; Lv, E ; Ci, Chunzeng ; Li, Xiangling

BMC nephrology, 2022-07, Vol.23 (1) [Periódico revisado por pares]

BioMed Central Ltd

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10
1,25-dihydroxyvitamin D3 ameliorates lupus nephritis through inhibiting the NF-κB and MAPK signalling pathways in MRL/lpr mice
1,25-dihydroxyvitamin D3 ameliorates lupus nephritis through inhibiting the NF-κB and MAPK signalling pathways in MRL/lpr mice
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1,25-dihydroxyvitamin D3 ameliorates lupus nephritis through inhibiting the NF-κB and MAPK signalling pathways in MRL/lpr mice

Li, Xuewei ; Liu, Jie ; Zhao, Yingzhe ; Xu, Ning ; Lv, E. ; Ci, Chunzeng ; Li, Xiangling

BMC nephrology, 2022-07, Vol.23 (1), p.1-243, Article 243 [Periódico revisado por pares]

London: BioMed Central

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