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Refinado por: assunto: Localization remover BACON - Mir@bel - GLOBAL_LIBRESACCES remover
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1
ABA represses TOR and root meristem activity through nuclear exit of the SnRK1 kinase
ABA represses TOR and root meristem activity through nuclear exit of the SnRK1 kinase
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ABA represses TOR and root meristem activity through nuclear exit of the SnRK1 kinase

Belda-Palazón, Borja ; Costa, Mónica ; Beeckman, Tom ; Rolland, Filip ; Baena-González, Elena

Proceedings of the National Academy of Sciences - PNAS, 2022-07, Vol.119 (28), p.1-e2204862119 [Periódico revisado por pares]

Washington: National Academy of Sciences

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2
Aberrant Upregulation of Indoleamine 2,3-Dioxygenase 1 Promotes Proliferation and Metastasis of Hepatocellular Carcinoma Cells via Coordinated Activation of AhR and β-Catenin Signaling
Aberrant Upregulation of Indoleamine 2,3-Dioxygenase 1 Promotes Proliferation and Metastasis of Hepatocellular Carcinoma Cells via Coordinated Activation of AhR and β-Catenin Signaling
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Aberrant Upregulation of Indoleamine 2,3-Dioxygenase 1 Promotes Proliferation and Metastasis of Hepatocellular Carcinoma Cells via Coordinated Activation of AhR and β-Catenin Signaling

Chen, Chih-Ta ; Wu, Pei-Hua ; Hu, Chia-Chi ; Nien, Hsiao-Ching ; Wang, Jin-Town ; Sheu, Jin-Chuan ; Chow, Lu-Ping

International journal of molecular sciences, 2021-10, Vol.22 (21), p.11661 [Periódico revisado por pares]

Switzerland: MDPI AG

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3
Abnormal Localization and Tumor Suppressor Function of Epithelial Tissue-Specific Transcription Factor ESE3 in Esophageal Squamous Cell Carcinoma
Abnormal Localization and Tumor Suppressor Function of Epithelial Tissue-Specific Transcription Factor ESE3 in Esophageal Squamous Cell Carcinoma
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Abnormal Localization and Tumor Suppressor Function of Epithelial Tissue-Specific Transcription Factor ESE3 in Esophageal Squamous Cell Carcinoma

Wang, Li ; Xing, Jie ; Cheng, Rui ; Shao, Ying ; Li, Peng ; Zhu, Shengtao ; Zhang, Shutian Mukaisho, Ken-ichi

PloS one, 2015-05, Vol.10 (5), p.e0126319 [Periódico revisado por pares]

United States: Public Library of Science

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4
Abnormal NFAT5 Physiology in Duchenne Muscular Dystrophy Fibroblasts as a Putative Explanation for the Permanent Fibrosis Formation in Duchenne Muscular Dystrophy
Abnormal NFAT5 Physiology in Duchenne Muscular Dystrophy Fibroblasts as a Putative Explanation for the Permanent Fibrosis Formation in Duchenne Muscular Dystrophy
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Abnormal NFAT5 Physiology in Duchenne Muscular Dystrophy Fibroblasts as a Putative Explanation for the Permanent Fibrosis Formation in Duchenne Muscular Dystrophy

Herbelet, Sandrine ; De Paepe, Boel ; De Bleecker, Jan L

International journal of molecular sciences, 2020-10, Vol.21 (21), p.7888 [Periódico revisado por pares]

Switzerland: MDPI AG

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5
ACADL-YAP axis activity in non-small cell lung cancer carcinogenicity
ACADL-YAP axis activity in non-small cell lung cancer carcinogenicity
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ACADL-YAP axis activity in non-small cell lung cancer carcinogenicity

Chen, Kegong ; Hong, Chunqiao ; Kong, Weibo ; Li, Guanghua ; Liu, Zhuang ; Zhu, Kechao ; Lu, Chen ; Si, Panpan ; Gao, Pan ; Ning, Guangyao ; Zhang, Renquan

Cancer Cell International, 2024-02, Vol.24 (1), p.86-86 [Periódico revisado por pares]

England: BioMed Central Ltd

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6
Acanthus ebracteatus leaf extract provides neuronal cell protection against oxidative stress injury induced by glutamate
Acanthus ebracteatus leaf extract provides neuronal cell protection against oxidative stress injury induced by glutamate
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Acanthus ebracteatus leaf extract provides neuronal cell protection against oxidative stress injury induced by glutamate

Prasansuklab, Anchalee ; Tencomnao, Tewin

BMC complementary and alternative medicine, 2018-10, Vol.18 (1), p.278-278, Article 278

England: BioMed Central Ltd

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7
Acetylation of conserved DVL-1 lysines regulates its nuclear translocation and binding to gene promoters in triple-negative breast cancer
Acetylation of conserved DVL-1 lysines regulates its nuclear translocation and binding to gene promoters in triple-negative breast cancer
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Acetylation of conserved DVL-1 lysines regulates its nuclear translocation and binding to gene promoters in triple-negative breast cancer

Sharma, Monica ; Molehin, Deborah ; Castro-Piedras, Isabel ; Martinez, Edgar G ; Pruitt, Kevin

Scientific reports, 2019-11, Vol.9 (1), p.16257-16, Article 16257 [Periódico revisado por pares]

England: Nature Publishing Group

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8
Acid ceramidase promotes nuclear export of PTEN through sphingosine 1-phosphate mediated Akt signaling
Acid ceramidase promotes nuclear export of PTEN through sphingosine 1-phosphate mediated Akt signaling
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Acid ceramidase promotes nuclear export of PTEN through sphingosine 1-phosphate mediated Akt signaling

Beckham, Thomas H ; Cheng, Joseph C ; Lu, Ping ; Marrison, S Tucker ; Norris, James S ; Liu, Xiang Zi, Xiaolin

PloS one, 2013-10, Vol.8 (10), p.e76593-e76593 [Periódico revisado por pares]

United States: Public Library of Science

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9
ACLY Nuclear Translocation in Human Macrophages Drives Proinflammatory Gene Expression by NF-κB Acetylation
ACLY Nuclear Translocation in Human Macrophages Drives Proinflammatory Gene Expression by NF-κB Acetylation
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ACLY Nuclear Translocation in Human Macrophages Drives Proinflammatory Gene Expression by NF-κB Acetylation

Santarsiero, Anna ; Convertini, Paolo ; Todisco, Simona ; Pierri, Ciro L ; De Grassi, Anna ; Williams, Niamh C ; Iacobazzi, Dominga ; De Stefano, Giulio ; O'Neill, Luke A J ; Infantino, Vittoria

Cells (Basel, Switzerland), 2021-10, Vol.10 (11), p.2962 [Periódico revisado por pares]

Switzerland: MDPI AG

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10
Actin polymerization negatively regulates p53 function by impairing its nuclear import in response to DNA damage
Actin polymerization negatively regulates p53 function by impairing its nuclear import in response to DNA damage
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Actin polymerization negatively regulates p53 function by impairing its nuclear import in response to DNA damage

Wang, Ling ; Wang, Min ; Wang, Shuyan ; Qi, Tianyang ; Guo, Lijing ; Li, Jinjiao ; Qi, Wenjing ; Ampah, Khamal Kwesi ; Ba, Xueqing ; Zeng, Xianlu Prigent, Claude

PloS one, 2013-04, Vol.8 (4), p.e60179-e60179 [Periódico revisado por pares]

United States: Public Library of Science

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