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Refinado por: Nome da Publicação: Brain Research remover assunto: Central Neurotransmission. Neuromudulation. Pathways And Receptors remover
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1
Knockout of the γ-aminobutyric acid receptor subunit α4 reduces functional δ-containing extrasynaptic receptors in hippocampal pyramidal cells at the onset of puberty
Knockout of the γ-aminobutyric acid receptor subunit α4 reduces functional δ-containing extrasynaptic receptors in hippocampal pyramidal cells at the onset of puberty
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Knockout of the γ-aminobutyric acid receptor subunit α4 reduces functional δ-containing extrasynaptic receptors in hippocampal pyramidal cells at the onset of puberty

Sabaliauskas, Nicole ; Shen, Hui ; Homanics, Gregg E ; Smith, Sheryl S ; Aoki, Chiye

Brain research, 2012-04, Vol.1450, p.11-23 [Periódico revisado por pares]

Amsterdam: Elsevier B.V

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2
γ-Aminobutyric acid receptor subtype antagonists differentially alter opioid-induced feeding in the shell region of the nucleus accumbens in rats
γ-Aminobutyric acid receptor subtype antagonists differentially alter opioid-induced feeding in the shell region of the nucleus accumbens in rats
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γ-Aminobutyric acid receptor subtype antagonists differentially alter opioid-induced feeding in the shell region of the nucleus accumbens in rats

Znamensky, Vladimir ; Echo, Joyce A ; Lamonte, Nicole ; Christian, Garrison ; Ragnauth, André ; Bodnar, Richard J

Brain research, 2001-07, Vol.906 (1), p.84-91 [Periódico revisado por pares]

London: Elsevier B.V

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3
Distribution of 5-HT2A receptor immunoreactivity in the rat amygdaloid complex and colocalization with γ-aminobutyric acid
Distribution of 5-HT2A receptor immunoreactivity in the rat amygdaloid complex and colocalization with γ-aminobutyric acid
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Distribution of 5-HT2A receptor immunoreactivity in the rat amygdaloid complex and colocalization with γ-aminobutyric acid

Bombardi, Cristiano

Brain research, 2011-01, Vol.1370, p.112-128 [Periódico revisado por pares]

Amsterdam: Elsevier B.V

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4
Immunohistochemical studies on distribution of GABAA receptor complex in the rat brain using antibody against purified GABAA receptor complex
Immunohistochemical studies on distribution of GABAA receptor complex in the rat brain using antibody against purified GABAA receptor complex
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Immunohistochemical studies on distribution of GABAA receptor complex in the rat brain using antibody against purified GABAA receptor complex

TAGUCHI, J ; KURIYAMA, T ; OHMORI, Y ; KURIYAMA, K

Brain research, 1989-04, Vol.483 (2), p.395-401 [Periódico revisado por pares]

London: Elsevier

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5
Chronic exposure of developing cortical neurons to GABA down-regulates GABA/benzodiazepine receptors and GABA-gated chloride currents
Chronic exposure of developing cortical neurons to GABA down-regulates GABA/benzodiazepine receptors and GABA-gated chloride currents
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Chronic exposure of developing cortical neurons to GABA down-regulates GABA/benzodiazepine receptors and GABA-gated chloride currents

Hablitz, John J. ; Jalilian Tehrani, Mohammad H. ; Barnes, Eugene M.

Brain research, 1989-11, Vol.501 (2), p.332-338 [Periódico revisado por pares]

London: Elsevier B.V

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6
Genetic selection for benzodiazepine ataxia produces functional changes in the γ-aminobutyric acid receptor chloride channel complex
Genetic selection for benzodiazepine ataxia produces functional changes in the γ-aminobutyric acid receptor chloride channel complex
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Genetic selection for benzodiazepine ataxia produces functional changes in the γ-aminobutyric acid receptor chloride channel complex

Allan, Andrea M. ; Gallaher, Edward J. ; Gionet, Susanne E. ; Harris, R. Adron

Brain research, 1988-06, Vol.452 (1), p.118-126 [Periódico revisado por pares]

London: Elsevier B.V

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7
γ-aminobutyric acid, through GABAA receptors, inhibits the potassium-stimulated release of calcitonin gene-related peptide-but not that of substance P-like material from rat spinal cord slices
γ-aminobutyric acid, through GABAA receptors, inhibits the potassium-stimulated release of calcitonin gene-related peptide-but not that of substance P-like material from rat spinal cord slices
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γ-aminobutyric acid, through GABAA receptors, inhibits the potassium-stimulated release of calcitonin gene-related peptide-but not that of substance P-like material from rat spinal cord slices

BOURGOIN, S ; POHL, M ; BENOLIEL, J. J ; MAUBORGNE, A ; COLLIN, E ; HAMON, M ; CESSELIN, F

Brain research, 1992-06, Vol.583 (1-2), p.344-348 [Periódico revisado por pares]

London: Elsevier

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8
Complex interactions between pregnenolone sulfate and the t-butylbicyclophosphorothionate-labeled chloride ionophore in rat brain
Complex interactions between pregnenolone sulfate and the t-butylbicyclophosphorothionate-labeled chloride ionophore in rat brain
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Complex interactions between pregnenolone sulfate and the t-butylbicyclophosphorothionate-labeled chloride ionophore in rat brain

Gee, Kelvin W. ; Joy, Darrin S. ; Belelli, Delia

Brain research, 1989-03, Vol.482 (1), p.169-173 [Periódico revisado por pares]

London: Elsevier B.V

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9
Evidence for the independent role of GABA synapses of the zona incerta-lateral hypothalamic region in haloperidol-induced catalepsy
Evidence for the independent role of GABA synapses of the zona incerta-lateral hypothalamic region in haloperidol-induced catalepsy
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Evidence for the independent role of GABA synapses of the zona incerta-lateral hypothalamic region in haloperidol-induced catalepsy

Wardas, Jadwiga ; Ossowska, Krystyna ; Wolfarth, Stanisław

Brain research, 1988-10, Vol.462 (2), p.378-382 [Periódico revisado por pares]

London: Elsevier B.V

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10
Changes in γ-aminobutyric acid, μ-opioid and neurotensin receptors in the accumbens-pallidal projection after discrete quinolinic acid lesions in the nucleus accumbens
Changes in γ-aminobutyric acid, μ-opioid and neurotensin receptors in the accumbens-pallidal projection after discrete quinolinic acid lesions in the nucleus accumbens
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Changes in γ-aminobutyric acid, μ-opioid and neurotensin receptors in the accumbens-pallidal projection after discrete quinolinic acid lesions in the nucleus accumbens

Churchill, Lynn ; Dilts, Roger P. ; Kalivas, Peter W.

Brain research, 1990-03, Vol.511 (1), p.41-54 [Periódico revisado por pares]

London: Elsevier B.V

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