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1
Methyl-CpG binding protein 2 is associated with the prognosis and mortality of elderly patients with hip fractures
Methyl-CpG binding protein 2 is associated with the prognosis and mortality of elderly patients with hip fractures
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Methyl-CpG binding protein 2 is associated with the prognosis and mortality of elderly patients with hip fractures

Gao, Xuejian; Xue, Shan; Yang, Fuqiang; Wu, Baoling; Yu, Xiaojing; An, Baoquan

Clinics; v. 77 (2022); 100034

Hospital das Clínicas, Faculdade de Medicina, Universidade de São Paulo 2022-04-15

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2
Antioxidant and anti-inflammatory properties of alpha-lipoic acid protect against valproic acid–induced liver injury
Antioxidant and anti-inflammatory properties of alpha-lipoic acid protect against valproic acid–induced liver injury
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Antioxidant and anti-inflammatory properties of alpha-lipoic acid protect against valproic acid–induced liver injury

Mohammed, Marwa Abdeltawab ; Gharib, Doaa Mostafa ; Reyad, Hoda Ramadan ; Mohamed, Alaa Aboud ; Elroby, Fadwa A ; Mahmoud, Hoda Sayed

Canadian journal of physiology and pharmacology, 2021-05, Vol.99 (5), p.499-505 [Periódico revisado por pares]

1840 Woodward Drive, Suite 1, Ottawa, ON K2C 0P7: NRC Research Press

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3
Protective Role of Smad6 in Inflammation-Induced Valvular Cell Calcification
Protective Role of Smad6 in Inflammation-Induced Valvular Cell Calcification
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Protective Role of Smad6 in Inflammation-Induced Valvular Cell Calcification

Li, Xin ; Lim, Jina ; Lu, Jinxiu ; Pedego, Taylor M ; Demer, Linda ; Tintut, Yin

Journal of cellular biochemistry, 2015-10, Vol.116 (10), p.2354-2364 [Periódico revisado por pares]

United States: Blackwell Publishing Ltd

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4
Meta-analysis of the Efficacy and Safety of Adalimumab, Etanercept, and Infliximab for the Treatment of Rheumatoid Arthritis
Meta-analysis of the Efficacy and Safety of Adalimumab, Etanercept, and Infliximab for the Treatment of Rheumatoid Arthritis
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Meta-analysis of the Efficacy and Safety of Adalimumab, Etanercept, and Infliximab for the Treatment of Rheumatoid Arthritis

Wiens, Astrid ; Venson, Rafael ; Correr, Cassyano J. ; Otuki, Michel Fleith ; Pontarolo, Roberto

Pharmacotherapy, 2010-04, Vol.30 (4), p.339-353 [Periódico revisado por pares]

Oxford, UK: Blackwell Publishing Ltd

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5
Berberine protects endothelial progenitor cell from damage of TNF-α via the PI3K/AKT/eNOS signaling pathway
Berberine protects endothelial progenitor cell from damage of TNF-α via the PI3K/AKT/eNOS signaling pathway
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Berberine protects endothelial progenitor cell from damage of TNF-α via the PI3K/AKT/eNOS signaling pathway

Xiao, Min ; Men, Li Na ; Xu, Ming Guo ; Wang, Guo Bing ; Lv, Hai Tao ; Liu, Cong

European journal of pharmacology, 2014-11, Vol.743, p.11-16 [Periódico revisado por pares]

Netherlands: Elsevier B.V

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6
Mechanism of the inhibitory effect of melatonin on tumor necrosis factor production in vivo and in vitro
Mechanism of the inhibitory effect of melatonin on tumor necrosis factor production in vivo and in vitro
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Mechanism of the inhibitory effect of melatonin on tumor necrosis factor production in vivo and in vitro

Sacco, Silvano ; Aquilini, Luciano ; Ghezzi, Pietro ; Pinza, Mario ; Guglielmotti, Angelo

European journal of pharmacology, 1998-02, Vol.343 (2), p.249-255 [Periódico revisado por pares]

Amsterdam: Elsevier B.V

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7
Tumour necrosis factor α signalling through activation of Kupffer cells plays an essential role in liver fibrosis of non-alcoholic steatohepatitis in mice
Tumour necrosis factor α signalling through activation of Kupffer cells plays an essential role in liver fibrosis of non-alcoholic steatohepatitis in mice
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Tumour necrosis factor α signalling through activation of Kupffer cells plays an essential role in liver fibrosis of non-alcoholic steatohepatitis in mice

Tomita, K ; Tamiya, G ; Ando, S ; Ohsumi, K ; Chiyo, T ; Mizutani, A ; Kitamura, N ; Toda, K ; Kaneko, T ; Horie, Y ; Han, J-Y ; Kato, S ; Shimoda, M ; Oike, Y ; Tomizawa, M ; Makino, S ; Ohkura, T ; Saito, H ; Kumagai, N ; Nagata, H ; Ishii, H ; Hibi, T

Gut, 2006-03, Vol.55 (3), p.415-424 [Periódico revisado por pares]

London: BMJ Publishing Group Ltd and British Society of Gastroenterology

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8
Interactions and Trends of Interleukins, PAI-1, CRP, and TNF-α in Inflammatory Responses during the Perioperative Period of Joint Arthroplasty: Implications for Pain Management-A Narrative Review
Interactions and Trends of Interleukins, PAI-1, CRP, and TNF-α in Inflammatory Responses during the Perioperative Period of Joint Arthroplasty: Implications for Pain Management-A Narrative Review
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Interactions and Trends of Interleukins, PAI-1, CRP, and TNF-α in Inflammatory Responses during the Perioperative Period of Joint Arthroplasty: Implications for Pain Management-A Narrative Review

Cocea, Arabela-Codruta ; Stoica, Cristian Ioan

Journal of personalized medicine, 2024-05, Vol.14 (5), p.537 [Periódico revisado por pares]

Switzerland: MDPI AG

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9
Anticancer Tetrahydrocarbazoles: A Wide Journey from 2000 Till Date
Anticancer Tetrahydrocarbazoles: A Wide Journey from 2000 Till Date
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Anticancer Tetrahydrocarbazoles: A Wide Journey from 2000 Till Date

Kumar, Nitin ; Gupta, Pankaj

Letters in drug design & discovery, 2024-03, Vol.21 (3), p.421-439 [Periódico revisado por pares]

Sharjah: Benham Science Publishers

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10
Interleukin-1β but not tumor necrosis factor-α potentiates neuronal damage by quinolinic acid: Protection by an adenosine A2A receptor antagonist
Interleukin-1β but not tumor necrosis factor-α potentiates neuronal damage by quinolinic acid: Protection by an adenosine A2A receptor antagonist
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Interleukin-1β but not tumor necrosis factor-α potentiates neuronal damage by quinolinic acid: Protection by an adenosine A2A receptor antagonist

Stone, Trevor W. ; Behan, Wilhelmina M.H.

Journal of neuroscience research, 2007-04, Vol.85 (5), p.1077-1085 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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Deste Autor:

  1. Gao, X
  2. An, B
  3. Wu, B
  4. Yang, F
  5. Xue, S

Neste Assunto:

  1. Tnf-Α
  2. Tnf-Α
  3. Science & Technology
  4. Life Sciences & Biomedicine
  5. Tumor Necrosis Factor-Α

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