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Refinado por: autor: Vijay-Kumar, Matam remover
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1
Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5
Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5
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Metabolic Syndrome and Altered Gut Microbiota in Mice Lacking Toll-Like Receptor 5

Vijay-Kumar, Matam ; Aitken, Jesse D ; Carvalho, Frederic A ; Cullender, Tyler C ; Mwangi, Simon ; Srinivasan, Shanthi ; Sitaraman, Shanthi V ; Knight, Rob ; Ley, Ruth E ; Gewirtz, Andrew T

Science (American Association for the Advancement of Science), 2010-04, Vol.328 (5975), p.228-231 [Periódico revisado por pares]

Washington, DC: American Association for the Advancement of Science

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2
Toll-like receptor-gut microbiota interactions: perturb at your own risk
Toll-like receptor-gut microbiota interactions: perturb at your own risk
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Toll-like receptor-gut microbiota interactions: perturb at your own risk

Carvalho, Frederic A ; Aitken, Jesse D ; Vijay-Kumar, Matam ; Gewirtz, Andrew T

Annual review of physiology, 2012-01, Vol.74 (1), p.177-198 [Periódico revisado por pares]

United States: Annual Reviews, Inc

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3
Lipocalin 2 deficiency dysregulates iron homeostasis and exacerbates endotoxin-induced sepsis
Lipocalin 2 deficiency dysregulates iron homeostasis and exacerbates endotoxin-induced sepsis
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Lipocalin 2 deficiency dysregulates iron homeostasis and exacerbates endotoxin-induced sepsis

Srinivasan, Gayathri ; Aitken, Jesse D ; Zhang, Benyue ; Carvalho, Frederic A ; Chassaing, Benoit ; Shashidharamurthy, Rangaiah ; Borregaard, Niels ; Jones, Dean P ; Gewirtz, Andrew T ; Vijay-Kumar, Matam

The Journal of immunology (1950), 2012-08, Vol.189 (4), p.1911-1919 [Periódico revisado por pares]

United States: Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists

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4
Transient Inability to Manage Proteobacteria Promotes Chronic Gut Inflammation in TLR5-Deficient Mice
Transient Inability to Manage Proteobacteria Promotes Chronic Gut Inflammation in TLR5-Deficient Mice
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Transient Inability to Manage Proteobacteria Promotes Chronic Gut Inflammation in TLR5-Deficient Mice

Carvalho, Frederic A. ; Koren, Omry ; Goodrich, Julia K. ; Johansson, Malin E.V. ; Nalbantoglu, Ilke ; Aitken, Jesse D. ; Su, Yueju ; Chassaing, Benoit ; Walters, William A. ; González, Antonio ; Clemente, Jose C. ; Cullender, Tyler C. ; Barnich, Nicolas ; Darfeuille-Michaud, Arlette ; Vijay-Kumar, Matam ; Knight, Rob ; Ley, Ruth E. ; Gewirtz, Andrew T.

Cell host & microbe, 2012-08, Vol.12 (2), p.139-152 [Periódico revisado por pares]

United States: Elsevier Inc

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5
TLR5 or NLRC4 is necessary and sufficient for promotion of humoral immunity by flagellin
TLR5 or NLRC4 is necessary and sufficient for promotion of humoral immunity by flagellin
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TLR5 or NLRC4 is necessary and sufficient for promotion of humoral immunity by flagellin

Vijay‐Kumar, Matam ; Carvalho, Frederic A ; Aitken, Jesse D ; Fifadara, Nimita H ; Gewirtz, Andrew T

European journal of immunology, 2010-12, Vol.40 (12), p.3528-3534 [Periódico revisado por pares]

Weinheim: Wiley‐VCH Verlag

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6
Cytosolic flagellin receptor NLRC4 protects mice against mucosal and systemic challenges
Cytosolic flagellin receptor NLRC4 protects mice against mucosal and systemic challenges
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Cytosolic flagellin receptor NLRC4 protects mice against mucosal and systemic challenges

Carvalho, F A ; Nalbantoglu, I ; Aitken, J D ; Uchiyama, R ; Su, Y ; Doho, G H ; Vijay-Kumar, M ; Gewirtz, A T

Mucosal immunology, 2012-05, Vol.5 (3), p.288-298 [Periódico revisado por pares]

United States: Nature Publishing Group

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7
TLR5 activation induces secretory interleukin-1 receptor antagonist (sIL-1Ra) and reduces inflammasome-associated tissue damage
TLR5 activation induces secretory interleukin-1 receptor antagonist (sIL-1Ra) and reduces inflammasome-associated tissue damage
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TLR5 activation induces secretory interleukin-1 receptor antagonist (sIL-1Ra) and reduces inflammasome-associated tissue damage

Carvalho, F A ; Aitken, J D ; Gewirtz, A T ; Vijay-Kumar, M

Mucosal immunology, 2011-01, Vol.4 (1), p.102-111 [Periódico revisado por pares]

United States: Nature Publishing Group

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8
Intestinal epithelia activate anti-viral signaling via intracellular sensing of rotavirus structural components
Intestinal epithelia activate anti-viral signaling via intracellular sensing of rotavirus structural components
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Intestinal epithelia activate anti-viral signaling via intracellular sensing of rotavirus structural components

Frias, A H ; Vijay-Kumar, M ; Gentsch, J R ; Crawford, S E ; Carvalho, F A ; Estes, M K ; Gewirtz, A T

Mucosal immunology, 2010-11, Vol.3 (6), p.622-632 [Periódico revisado por pares]

United States: Nature Publishing Group

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9
Interleukin-1β (IL-1β) promotes susceptibility of Toll-like receptor 5 (TLR5) deficient mice to colitis
Interleukin-1β (IL-1β) promotes susceptibility of Toll-like receptor 5 (TLR5) deficient mice to colitis
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Interleukin-1β (IL-1β) promotes susceptibility of Toll-like receptor 5 (TLR5) deficient mice to colitis

Carvalho, Frederic A ; Nalbantoglu, Ilke ; Ortega-Fernandez, Sophie ; Aitken, Jesse D ; Su, Yueju ; Koren, Omry ; Walters, William A ; Knight, Rob ; Ley, Ruth E ; Vijay-Kumar, Matam ; Gewirtz, Andrew T

Gut, 2012-03, Vol.61 (3), p.373-384 [Periódico revisado por pares]

London: BMJ Publishing Group Ltd and British Society of Gastroenterology

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10
Loss of function mutation in toll-like receptor-4 does not offer protection against obesity and insulin resistance induced by a diet high in trans fat in mice
Loss of function mutation in toll-like receptor-4 does not offer protection against obesity and insulin resistance induced by a diet high in trans fat in mice
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Loss of function mutation in toll-like receptor-4 does not offer protection against obesity and insulin resistance induced by a diet high in trans fat in mice

Vijay-Kumar, Matam ; Aitken, Jesse D ; Carvalho, Frederic A ; Ziegler, Thomas R ; Gewirtz, Andrew T ; Ganji, Vijay

Journal of inflammation (London, England), 2011-02, Vol.8 (1), p.2-2, Article 2 [Periódico revisado por pares]

England: BioMed Central Ltd

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