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Refinado por: assunto: Jnk Mitogen-Activated Protein Kinases remover
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1
Glutathione S-transferase P1–1 (GSTP1–1) Inhibits c-Jun N-terminal Kinase (JNK1) Signaling through Interaction with the C Terminus
Glutathione S-transferase P1–1 (GSTP1–1) Inhibits c-Jun N-terminal Kinase (JNK1) Signaling through Interaction with the C Terminus
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Glutathione S-transferase P1–1 (GSTP1–1) Inhibits c-Jun N-terminal Kinase (JNK1) Signaling through Interaction with the C Terminus

Wang, Tieli ; Arifoglu, Pinar ; Ronai, Ze'ev ; Tew, Kenneth D.

The Journal of biological chemistry, 2001-06, Vol.276 (24), p.20999-21003 [Periódico revisado por pares]

United States: Elsevier Inc

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2
Conformation-Dependent Phosphorylation of p53
Conformation-Dependent Phosphorylation of p53
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Conformation-Dependent Phosphorylation of p53

Adler, Victor ; Pincus, Matthew R. ; Minamoto, Toshinari ; Fuchs, Serge Y. ; Bluth, Mark J. ; Brand-Rauf, Paul W. ; Friedman, Fred K. ; Robinson, Richard C. ; Chen, James M. ; Wang, Xin Wei ; Harris, Curtis C. ; Ze'ev Ronai

Proceedings of the National Academy of Sciences - PNAS, 1997-03, Vol.94 (5), p.1686-1691 [Periódico revisado por pares]

United States: National Academy of Sciences of the United States of America

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3
The Cytoprotective Aminothiol WR1065 Activates p53 through a Non-genotoxic Signaling Pathway Involving c-Jun N-terminal Kinase
The Cytoprotective Aminothiol WR1065 Activates p53 through a Non-genotoxic Signaling Pathway Involving c-Jun N-terminal Kinase
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The Cytoprotective Aminothiol WR1065 Activates p53 through a Non-genotoxic Signaling Pathway Involving c-Jun N-terminal Kinase

Pluquet, Olivier ; North, Sophie ; Bhoumik, Anindita ; Dimas, Konstantinos ; Ronai, Ze'ev ; Hainaut, Pierre

The Journal of biological chemistry, 2003-04, Vol.278 (14), p.11879-11887 [Periódico revisado por pares]

United States: American Society for Biochemistry and Molecular Biology

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4
Effects of chemopreventive selenium compounds on Jun N-kinase activities
Effects of chemopreventive selenium compounds on Jun N-kinase activities
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Effects of chemopreventive selenium compounds on Jun N-kinase activities

Adler, Victor ; Pincus, Matthew R. ; Posner, Scott ; Upadhyaya, Pramod ; El-Bayoumy, Karam ; Ronai, Ze'ev

Carcinogenesis (New York), 1996-09, Vol.17 (9), p.1849-1854 [Periódico revisado por pares]

Oxford: Oxford University Press

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5
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway
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Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway

Chie, L ; Amar, S ; Kung, H F ; Lin, M C ; Chen, H ; Chung, D L ; Adler, V ; Ronai, Z ; Friedman, F K ; Robinson, R C ; Kovac, C ; Brandt-Rauf, P W ; Yamaizumi, Z ; Michl, J ; Pincus, M R

Cancer chemotherapy and pharmacology, 2000, Vol.45 (6), p.441-449 [Periódico revisado por pares]

Germany

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6
Glutathione-S-transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase
Glutathione-S-transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase
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Glutathione-S-transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase

VILLAFANIA, A ; ANWAR, K ; KUNG, H.-F ; PINCUS, M. R ; AMAR, S ; CHIE, L ; WAY, D ; CHUNG, D. L ; ADLER, V ; RONAI, Z ; BRANDT-RAUF, P. W ; YAMAIZUMII, Z

Annals of clinical and laboratory science, 2000, Vol.30 (1), p.57-64 [Periódico revisado por pares]

Philadelphia, PA: Institute for Clinical Science

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7
Induction of beta-transducin repeat-containing protein by JNK signaling and its role in the activation of NF-kappaB
Induction of beta-transducin repeat-containing protein by JNK signaling and its role in the activation of NF-kappaB
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Induction of beta-transducin repeat-containing protein by JNK signaling and its role in the activation of NF-kappaB

Spiegelman, V S ; Stavropoulos, P ; Latres, E ; Pagano, M ; Ronai, Z ; Slaga, T J ; Fuchs, S Y

The Journal of biological chemistry, 2001-07, Vol.276 (29), p.27152-27158 [Periódico revisado por pares]

United States

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8
Selective inhibition of oncogenic ras-p21 in vivo by agents that block its interaction with jun-N-kinase (JNK) and jun proteins. Implications for the design of selective chemotherapeutic agents
Selective inhibition of oncogenic ras-p21 in vivo by agents that block its interaction with jun-N-kinase (JNK) and jun proteins. Implications for the design of selective chemotherapeutic agents
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Selective inhibition of oncogenic ras-p21 in vivo by agents that block its interaction with jun-N-kinase (JNK) and jun proteins. Implications for the design of selective chemotherapeutic agents

AMAR, S ; GLOZMAN, A ; CHUNG, D ; ADLER, V ; RONAI, Z ; FRIEDMAN, F. K ; ROBINSON, R ; BRANDT-RAUF, P ; YAMAIZUMI, Z ; PINCUS, M. R

Cancer chemotherapy and pharmacology, 1997, Vol.41 (1), p.79-85 [Periódico revisado por pares]

Berlin: Springer

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9
Complexes of p21RAS with JUN N-terminal kinase and JUN proteins
Complexes of p21RAS with JUN N-terminal kinase and JUN proteins
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Complexes of p21RAS with JUN N-terminal kinase and JUN proteins

Adler, V ; Pincus, M R ; Brandt-Rauf, P W ; Ronai, Z

Proceedings of the National Academy of Sciences - PNAS, 1995-11, Vol.92 (23), p.10585-10589 [Periódico revisado por pares]

United States: National Acad Sciences

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10
jun-NH2-terminal kinase activation mediated by UV-induced DNA lesions in melanoma and fibroblast cells
jun-NH2-terminal kinase activation mediated by UV-induced DNA lesions in melanoma and fibroblast cells
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jun-NH2-terminal kinase activation mediated by UV-induced DNA lesions in melanoma and fibroblast cells

ADLER, V ; FUCHS, S. Y ; KIM, J ; KRAFT, A ; KING, M. P ; PELLING, J ; RONAI, Z

Cell growth & differentiation, 1995-11, Vol.6 (11), p.1437-1446

Philadelphia, PA: American Association for Cancer Research

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