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Interaction between the HPV-16 E2 transcriptional activator and p53
Interaction between the HPV-16 E2 transcriptional activator and p53
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Interaction between the HPV-16 E2 transcriptional activator and p53

MASSIMI, P ; PIM, D ; BERTOLI, C ; BOUVARD, V ; BANKS, L

Oncogene, 1999-12, Vol.18 (54), p.7748-7754 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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2
Alternatively spliced HPV-18 E6 protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth
Alternatively spliced HPV-18 E6 protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth
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Alternatively spliced HPV-18 E6 protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth

PIM, D ; MASSIMI, P ; BANKS, L

Oncogene, 1997-07, Vol.15 (3), p.257-264 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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3
Alternatively spliced HPV-18 E6 super() protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth
Alternatively spliced HPV-18 E6 super() protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth
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Alternatively spliced HPV-18 E6 super() protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth

Pim, D ; Massimi, P ; Banks, L

Oncogene, 1997-07, Vol.15 (3), p.257-264 [Periódico revisado por pares]

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4
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A
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Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A

PIM, David ; MASSIMI, Paola ; DILWORTH, Stephen M ; BANKS, Lawrence

Oncogene, 2005-11, Vol.24 (53), p.7830-7838 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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5
Human papillomaviruses, cervical cancer and cell polarity
Human papillomaviruses, cervical cancer and cell polarity
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Human papillomaviruses, cervical cancer and cell polarity

Thomas, M ; Narayan, N ; Pim, D ; Tomaić, V ; Massimi, P ; Nagasaka, K ; Kranjec, C ; Gammoh, N ; Banks, L

Oncogene, 2008-11, Vol.27 (55), p.7018-7030 [Periódico revisado por pares]

England: Nature Publishing Group

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6
Epigenomic alterations define lethal CIMP-positive ependymomas of infancy
Epigenomic alterations define lethal CIMP-positive ependymomas of infancy
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Epigenomic alterations define lethal CIMP-positive ependymomas of infancy

Mack, S C ; Witt, H ; Piro, R M ; Gu, L ; Zuyderduyn, S ; Stütz, A M ; Wang, X ; Gallo, M ; Garzia, L ; Zayne, K ; Zhang, X ; Ramaswamy, V ; Jäger, N ; Jones, D T W ; Sill, M ; Pugh, T J ; Ryzhova, M ; Wani, K M ; Shih, D J H ; Head, R ; Remke, M ; Bailey, S D ; Zichner, T ; Faria, C C ; Barszczyk, M ; Stark, S ; Seker-Cin, H ; Hutter, S ; Johann, P ; Bender, S ; Hovestadt, V ; Tzaridis, T ; Dubuc, A M ; Northcott, P A ; Peacock, J ; Bertrand, K C ; Agnihotri, S ; Cavalli, F M G ; Clarke, I ; Nethery-Brokx, K ; Creasy, C L ; Verma, S K ; Koster, J ; Wu, X ; Yao, Y ; Milde, T ; Sin-Chan, P ; Zuccaro, J ; Lau, L ; Pereira, S ; Castelo-Branco, P ; Hirst, M ; Marra, M A ; Roberts, S S ; Fults, D ; Massimi, L ; Cho, Y J ; Van Meter, T ; Grajkowska, W ; Lach, B ; Kulozik, A E ; von Deimling, A ; Witt, O ; Scherer, S W ; Fan, X ; Muraszko, K M ; Kool, M ; Pomeroy, S L ; Gupta, N ; Phillips, J ; Huang, A ; Tabori, U ; Hawkins, C ; Malkin, D ; Kongkham, P N ; Weiss, W A ; Jabado, N ; Rutka, J T ; Bouffet, E ; Korbel, J O ; Lupien, M ; Aldape, K D ; Bader, G D ; Eils, R ; Lichter, P ; Dirks, P B ; Pfister, S M ; Korshunov, A ; Taylor, M D

Nature (London), 2014-02, Vol.506 (7489), p.445-450 [Periódico revisado por pares]

England: Nature Publishing Group

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7
The cell polarity regulator hScrib controls ERK activation through a KIM site-dependent interaction
The cell polarity regulator hScrib controls ERK activation through a KIM site-dependent interaction
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The cell polarity regulator hScrib controls ERK activation through a KIM site-dependent interaction

NAGASAKA, K ; PIM, D ; MYERS, M ; BANKS, L ; MASSIMI, P ; THOMAS, M ; TOMAIC, V ; SUBBAIAH, V. K ; KRANJEC, C ; NAKAGAWA, S ; YANO, T ; TAKETANI, Y

Oncogene, 2010-09, Vol.29 (38), p.5311-5321 [Periódico revisado por pares]

Basingstoke: Nature Publishing Group

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8
ADAR2-editing activity inhibits glioblastoma growth through the modulation of the CDC14B/Skp2/p21/p27 axis
ADAR2-editing activity inhibits glioblastoma growth through the modulation of the CDC14B/Skp2/p21/p27 axis
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ADAR2-editing activity inhibits glioblastoma growth through the modulation of the CDC14B/Skp2/p21/p27 axis

Galeano, F ; Rossetti, C ; Tomaselli, S ; Cifaldi, L ; Lezzerini, M ; Pezzullo, M ; Boldrini, R ; Massimi, L ; Di Rocco, C M ; Locatelli, F ; Gallo, A

Oncogene, 2013-02, Vol.32 (8), p.998-1009 [Periódico revisado por pares]

England: Nature Publishing Group

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9
The hScrib Dlg apico-basal control complex is differentially targeted by HPV-16 and HPV-18 E6 proteins
The hScrib Dlg apico-basal control complex is differentially targeted by HPV-16 and HPV-18 E6 proteins
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The hScrib Dlg apico-basal control complex is differentially targeted by HPV-16 and HPV-18 E6 proteins

THOMAS, Miranda ; MASSIMI, Paola ; NAVARRO, Christel ; BORG, Jean-Paul ; BANKS, Lawrence

Oncogene, 2005-09, Vol.24 (41), p.6222-6230 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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10
Human and primate tumour viruses use PDZ binding as an evolutionarily conserved mechanism of targeting cell polarity regulators
Human and primate tumour viruses use PDZ binding as an evolutionarily conserved mechanism of targeting cell polarity regulators
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Human and primate tumour viruses use PDZ binding as an evolutionarily conserved mechanism of targeting cell polarity regulators

TOMAI, V ; GARDIOL, D ; MASSIMI, P ; OZBUN, M ; MYERS, M ; BANKS, L

Oncogene, 2009-01, Vol.28 (1), p.1-8 [Periódico revisado por pares]

Basingstoke: Nature Publishing Group

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