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Novel insight into stem cell mobilization-Plasma sphingosine-1-phosphate is a major chemoattractant that directs the egress of hematopoietic stem progenitor cells from the bone marrow and its level in peripheral blood increases during mobilization due to activation of complement cascade membrane attack complex
Novel insight into stem cell mobilization-Plasma sphingosine-1-phosphate is a major chemoattractant that directs the egress of hematopoietic stem progenitor cells from the bone marrow and its level in peripheral blood increases during mobilization due to activation of complement cascade membrane attack complex
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Novel insight into stem cell mobilization-Plasma sphingosine-1-phosphate is a major chemoattractant that directs the egress of hematopoietic stem progenitor cells from the bone marrow and its level in peripheral blood increases during mobilization due to activation of complement cascade membrane attack complex

RATAJCZAK, M. Z ; LEE, H ; WYSOCZYNSKI, M ; WAN, W ; MARLICZ, W ; LAUGHLIN, M. J ; KUCIA, M ; JANOWSKA-WIECZOREK, A ; RATAJCZAK, J

Leukemia, 2010-05, Vol.24 (5), p.976-985 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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2
The bone marrow-expressed antimicrobial cationic peptide LL-37 enhances the responsiveness of hematopoietic stem progenitor cells to an SDF-1 gradient and accelerates their engraftment after transplantation
The bone marrow-expressed antimicrobial cationic peptide LL-37 enhances the responsiveness of hematopoietic stem progenitor cells to an SDF-1 gradient and accelerates their engraftment after transplantation
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The bone marrow-expressed antimicrobial cationic peptide LL-37 enhances the responsiveness of hematopoietic stem progenitor cells to an SDF-1 gradient and accelerates their engraftment after transplantation

WU, W ; KIM, C. H ; LIU, R ; KUCIA, M ; MARLICZ, W ; GRECO, N ; RATAJCZAK, J ; LAUGHLIN, M. J ; RATAJCZAK, M. Z

Leukemia, 2012-04, Vol.26 (4), p.736-745 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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3
Mobilization studies in complement-deficient mice reveal that optimal AMD3100 mobilization of hematopoietic stem cells depends on complement cascade activation by AMD3100-stimulated granulocytes
Mobilization studies in complement-deficient mice reveal that optimal AMD3100 mobilization of hematopoietic stem cells depends on complement cascade activation by AMD3100-stimulated granulocytes
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Mobilization studies in complement-deficient mice reveal that optimal AMD3100 mobilization of hematopoietic stem cells depends on complement cascade activation by AMD3100-stimulated granulocytes

LEE, H. M ; WYSOCZYNSKI, M ; LIU, R ; SHIN, D.-M ; KUCIA, M ; BETTO, M ; RATAJCZAK, J ; RATAJCZAK, M. Z

Leukemia, 2010-03, Vol.24 (3), p.573-582 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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4
Impaired mobilization of hematopoietic stem progenitor cells in C5-deficient mice supports the pivotal involvement of innate immunity in this process and reveals novel promobilization effects of granulocytes
Impaired mobilization of hematopoietic stem progenitor cells in C5-deficient mice supports the pivotal involvement of innate immunity in this process and reveals novel promobilization effects of granulocytes
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Impaired mobilization of hematopoietic stem progenitor cells in C5-deficient mice supports the pivotal involvement of innate immunity in this process and reveals novel promobilization effects of granulocytes

LEE, H. M ; WU, W ; WYSOCZYNSKI, M ; LIU, R ; ZUBA-SURMA, E. K ; KUCIA, M ; RATAJCZAK, J ; RATAJCZAK, M. Z

Leukemia, 2009-11, Vol.23 (11), p.2052-2062 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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5
Defective engraftment of C3aR hematopoietic stem progenitor cells shows a novel role of the C3a-C3aR axis in bone marrow homing
Defective engraftment of C3aR hematopoietic stem progenitor cells shows a novel role of the C3a-C3aR axis in bone marrow homing
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Defective engraftment of C3aR hematopoietic stem progenitor cells shows a novel role of the C3a-C3aR axis in bone marrow homing

WYSOCZYNSKI, M ; RECA, R ; LEE, H ; WU, W ; RATAJCZAK, J ; RATAJCZAK, M. Z

Leukemia, 2009-08, Vol.23 (8), p.1455-1461 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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6
A novel view of paroxysmal nocturnal hemoglobinuria pathogenesis: more motile PNH hematopoietic stem/progenitor cells displace normal HSPCs from their niches in bone marrow due to defective adhesion, enhanced migration and mobilization in response to erythrocyte-released sphingosine-1 phosphate gradient
A novel view of paroxysmal nocturnal hemoglobinuria pathogenesis: more motile PNH hematopoietic stem/progenitor cells displace normal HSPCs from their niches in bone marrow due to defective adhesion, enhanced migration and mobilization in response to erythrocyte-released sphingosine-1 phosphate gradient
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A novel view of paroxysmal nocturnal hemoglobinuria pathogenesis: more motile PNH hematopoietic stem/progenitor cells displace normal HSPCs from their niches in bone marrow due to defective adhesion, enhanced migration and mobilization in response to erythrocyte-released sphingosine-1 phosphate gradient

RATAJCZAK, J ; KUCIA, M ; MIERZEJEWSKA, K ; LIU, R ; KIM, C. H ; NATARAJAN, N ; SHARMA, V ; MILLER, D. M ; MACIEJEWSKI, J ; RATAJCZAK, M. Z

Leukemia, 2012-07, Vol.26 (7), p.1722-1725 [Periódico revisado por pares]

Avenel, NJ: Nature Publishing Group

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7
Bone Marrow Transplantation Temporarily Improves Pancreatic Function in Streptozotocin-Induced Diabetes: Potential Involvement of Very Small Embryonic-Like Cells
Bone Marrow Transplantation Temporarily Improves Pancreatic Function in Streptozotocin-Induced Diabetes: Potential Involvement of Very Small Embryonic-Like Cells
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Bone Marrow Transplantation Temporarily Improves Pancreatic Function in Streptozotocin-Induced Diabetes: Potential Involvement of Very Small Embryonic-Like Cells

YIMING HUANG ; KUCIA, Magda ; HUSSAIN, Lala-Rukh ; YUJIE WEN ; HONG XU ; JUN YAN ; RATAJCZAK, Mariusz Z ; ILDSTAD, Suzanne T

Transplantation, 2010-03, Vol.89 (6), p.677-685 [Periódico revisado por pares]

Hagerstown, MD: Lippincott Williams & Wilkins

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