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Refinado por: Base de dados/Biblioteca: Wiley-Blackwell Full Collection 2013 remover tipo de recurso: Reports remover
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A single somatic activating Asp578His mutation of the luteinizing hormone receptor causes Leydig cell tumour in boys with gonadotropin-independent precocious puberty
A single somatic activating Asp578His mutation of the luteinizing hormone receptor causes Leydig cell tumour in boys with gonadotropin-independent precocious puberty
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A single somatic activating Asp578His mutation of the luteinizing hormone receptor causes Leydig cell tumour in boys with gonadotropin-independent precocious puberty

d'Alva, Catarina B ; Brito, Vinicius N ; Palhares, Heloisa M C ; Carvalho, Filomena M ; Arnhold, Ivo J P ; Mendonca, Berenice B ; Latronico, Ana Claudia

Clinical endocrinology, 2006, Vol.65 (3), p.408-410

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Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocious puberty
Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocious puberty
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Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocious puberty

Latronico, Ana Claudia ; Billerbeck, Ana Elisa C ; Pinto, Emilia Modolo ; Brazil D'Alva, Catarina ; Arnhold, Ivo Jorge P ; Mendonca, Berenice B

Clinical endocrinology, 2003, Vol.59 (4), p.533-534

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